Traumatic Arterial Spasm
نویسندگان
چکیده
منابع مشابه
Traumatic Arterial Spasm
That the arteries undergo spasmodic contraction, when subjected to trauma or concussional violence, is now a well-recognized fact. Called by the French surgeons Stupeui Arterialleand by the German surgeons ' Kroh's Arterial Spasm', the phenomenon was described by Makins (1919) towards the end ot World War I. During World War II, several remarkable instances of the condition were cited from dive...
متن کاملDelayed arterial spasm and thrombosis as a cause of post-traumatic hemiplegia (spate thrombosis).
Delayed Arterial Spasm and Thrombosis as a Cause of Post-Traumatic Hemiplegia (Spate Thrombosis) • The case of a Marine in whom a vertex epidural hematoma developed, depressing the superior sagittal sinus as a result of a fracture of the vertex of the skull, is presented. The hematoma was removed, and the patient did well until signs of progressive right hemiparesis suddenly developed two days ...
متن کاملThe relief of traumatic arterial spasm in threatened Volkmann's ischaemic contracture.
The etiology of Volkmann’s contracture has been much debated. Incorrect bandaging (Volkmann 1881), venous occlusion (Murphy 1914, Brooks 1922, Jepson 1926), subfascial haematoma (Moulonguet and Sen#{232}que 1928, Jos#{233} Jorge 1925), arterial spasm (Griffiths 1940) are some of the causes that have been suggested. Leriche (1928) likened the changes in the muscles to those seen in cardiac and p...
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A Model for Experimental Cerebral A rterial Spasm • Vasospasm of the basilar artery of the dog was induced by subarachnoid injection of arterial blood through the cisterna magna. Cerebral angiography was employed to evaluate quantitative assessment of the spasm. Chronic vasospasm was successfully induced in 100% of surviving dogs. Biphasic vasoconstriction was observed. The acute phase occurred...
متن کاملPostinfarction Angina Caused by Coronary Arterial Spasm
Recurrent ST-segment elevations in leads where new Q waves developed were repeatedly recorded in six patients during a recovery phase of acute myocardial infarction. Such ST-segment elevations were transient, occurred with or without chest pain, and returned to control levels. No enzymatic changes signifying recurrent myocardial necrosis were found after each episode. Selective coronary cineang...
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ژورنال
عنوان ژورنال: BMJ
سال: 1943
ISSN: 0959-8138,1468-5833
DOI: 10.1136/bmj.2.4309.167